14.12 Treatment of venous insufficiency
Unlike occasionally experiencing heavy legs, venous insuciency is a result of organic damage to the
vein walls which clinically manifests as large or small varicose veins. These are the result of a permanent
dilation, secondary to the hyperpressure and stasis of the venous blood, to which is added progressive
hypoxia of the intima (inner layer of the wall).
The deficiency of the valves of the deep veins and the perforating veins is behind this process. Their role in
preventing the regurgitation of venous blood is no longer guaranteed. Hydrostatic pressure is accentuated
and muscle contractions are no longer sucient to evacuate the venous blood.
The blood stagnates and causes hyperpressure in the superficial veins until varicose distensions are
produced.
Stasis oedema is often associated with venous insuciency, but not always. Moreover, this oedema may
be present or absent in the same patient, depending on the time of day and how much time the patient
has spent standing up.
We must therefore distinguish between:
e. Venous insuciency without oedema.
f. Venous insuciency with oedema.
The implications for the type of the electrostimulation programme are dierent depending on whether
there is or is not an oedema associated with varicose veins.
The electrode placements for these programs are proposed with 4 stimulation channels (WIRELESS
PROFESSIONAL 4CH devices)
14.12.1 Venous insufficiency without oedema
On one hand, electrical stimulation must allow for an increase in the general blood flow (arterial as well as
venous) so as to improve the circulation of the interstitial fluid and increase oxygenation of the tissues and
the intima of the veins. On the other hand, it is necessary to drain the veins as much as possible to combat
stasis. The increase in arterial flow (and therefore capillary flow, and therefore venous flow) is achieved by
means of the optimum low frequency for increase of flow, i.e. 8 Hz.
The deep veins are drained by being compressed, which is caused by tetanic contractions of the leg
muscles. The programme therefore consists of short tetanic contractions of the leg muscles, separated by
long active pauses to increase the flow.
14.12.1.1 Protocol
Venous insuciency 1